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Each of the four Klow components has been studied in different preclinical models, and each is associated in published literature with a distinct mechanistic pathway rather than a shared effect

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These isoforms mitigate the mitochondrial ROS burden, a major source of oxidative stress in spermatozoa [66,67]

YAP-dependent necrosis occurs in early stages of Alzheimers disease and regulates mouse model pathology
