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It has been proposed that the coordinated control of the immune response in favor of Th2 cytokines, such as IL-3, IL-4, IL-5, IL-6, IL-9, IL-13, and GM-CSF, is caused by a decrease in the inhibitory influence of Th1 cytokines, particularly IL-18, IL-12, and IFN

These alterations are thought to impact gut barrier integrity and immune function, both of which are implicated in CFS/ME pathophysiology

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Glutathione disulfide levels rise, and oxygen consumption increases with platelet aggregation

While several recent studies have begun to dissect the role of individual interneuron subtypes in the regulation of learned fear, this mechanism likely does not contribute to the processing of anxiety information and will not be discussed further here