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When there is no hereditary component to the condition but high TMA excretion, symptoms similar to TMAU can manifest as secondary TMAU [3,6]

Some of the symptoms which characterize the inherited disease are mimicked by the administration of drugs interacting with OCTN2 as off-target or by some diet regimens (Pochini et al., 2009, 2019

L-carnitine treatment could not reverse the hypertriglyceridemia and hypercholesterolemia in the cachexic mice, and actually increased the serum levels of triglycerides and total cholesterol

ABCD1 exhibits highest affinities for very hydrophobic saturated very long-chain fatty acids (C24:0C26:0)

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However, most clinical studies of atherosclerotic diseases with dietary antioxidants have not generated clear results, partly because of the non-selective effects of these anti-oxidative drugs [7]