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Cyclodextrin Overcomes the Transport Defect in Nearly Every Organ of NPC1 Mice Leading to Excretion of Sequestered Cholesterol as Bile Acid

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It remains to be shown whether ROXY9 controls TGA1 activity through a direct redox mechanism or whether the highly solvent-exposed CCLC signature is important for redox-independent protein-protein interactions within regulatory complexes

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