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Initial infection events were typically spatially separated, allowing robust identification of secondary infections associated with cell-to-cell spread and division of infected host cells

While both Unfolded Protein Response (as evidenced by elevated Atf4 mRNA levels for example) and mTORC1-dependent signaling pathways were potentiated upon Stard7 deficiency, the oxidative phosphorylation (OXPHOS) as well as the cellular response to oxidative stress were impaired in IECs lacking Stard7 (Fig

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