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Moreover, BAs inhibit the activation of the NLRP3 inflammasome through the TGR5-cAMP- protein kinase A (PKA) axis, and block LPS-induced systemic inflammation as well as T2D-related inflammation [267, 268]

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Heinisch and Kirby, 2010), and a post-synaptic mode of action, possibly involving direct interactions of the receptor with GABA B receptors (Guyon et al., 2006)