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M.PothulaS.LiuR
[DOI] [PMC free article] [PubMed] [Google Scholar] Maciejewska D
Different cannabinoid analogs produced a wound-healing response in these models, which may have been mediated by the activation of CB1 and/or CB2 receptors, increasing anti-inflammatory factor synthesis, the indirect activation of TRPV1 and epidermal growth factor receptors, and the inhibition of the FAAH enzyme [140,142]
Biochem Soc Trans (2003) 31(6):13379
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